IFN-a Treatment Inhibits Acute Friend Retrovirus Replication Primarily through the Antiviral Effector Molecule Apobec3
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منابع مشابه
IFN-α treatment inhibits acute Friend retrovirus replication primarily through the antiviral effector molecule Apobec3.
Therapeutic administration of IFN-α in clinical trials significantly reduced HIV-1 plasma viral load and human T-lymphotropic virus type I proviral load in infected patients. The mechanism may involve the concerted action of multiple antiretroviral effectors collectively known as "restriction factors," which could vary in relative importance according to the magnitude of transcriptional inducti...
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تاریخ انتشار 2013